Biomedical Engineering Reference
In-Depth Information
90. Guo, D.Q., et al.: Tumor necrosis factor employs a protein-tyrosine phosphatase to inhibit
activation of KDR and vascular endothelial cell growth factor-induced endothelial cell
proliferation. J. Biol. Chem. 275(15), 11216-11221 (2000)
91. Huang, L., et al.: HCPTPA, a protein tyrosine phosphatase that regulates vascular
endothelial growth factor receptor-mediated signal transduction and biological activity.
J. Biol. Chem. 274(53), 38183-38188 (1999)
92. Huot, J., et al.: Oxidative stress-induced actin reorganization mediated by the p38 mitogen-
activated protein kinase/heat shock protein 27 pathway in vascular endothelial cells. Circ.
Res. 80(3), 383-392 (1997)
93. Ushio-Fukai, M., et al.: Reactive oxygen species mediate the activation of Akt/protein kinase B
by angiotensin II in vascular smooth muscle cells. J. Biol. Chem. 274(32), 22699-22704 (1999)
94. Wang, Y., et al.: The role of the NADPH oxidase complex, p38 MAPK, and Akt in regulating
human monocyte/macrophage survival. Am. J. Respir. Cell. Mol. Biol. 36(1), 68-77 (2007)
95. Jiang, F., Zhang, Y., Dusting, G.J.: NADPH oxidase-mediated redox signaling: roles in cellular
stress response, stress tolerance, and tissue repair. Pharmacol. Rev. 63(1), 218-242 (2011)
96. van Wetering, S., et al.: Reactive oxygen species mediate Rac-induced loss of cell-cell
adhesion in primary human endothelial cells. J. Cell Sci. 115(9), 1837-1846 (2002)
97. Moldovan, L., et al.: The actin cytoskeleton reorganization induced by Rac1 requires the
production of superoxide. Antioxid. Redox. Signal. 1(1), 29-43 (1999)
98. Armstrong, D., Sohal, R.S, Cutler, R.G.: Free Radicals in Molecular Biology, Aging, and
Disease. Lippincott-Raven, Philadelphia (1984)
99. Dimmeler, S., Zeiher, A.M.: Reactive oxygen species and vascular cell apoptosis in response
to angiotensin II and pro-atherosclerotic factors. Regul. Pept. 90(1-3), 19-25 (2000)
100. Marui, N., et al.: Vascular cell adhesion molecule-1 (VCAM-1) gene transcription and
expression are regulated through an antioxidant-sensitive mechanism in human vascular
endothelial cells. J. Clin. Invest. 92(4), 1866-1874 (1993)
101. Chappell, D.C., et al.: Oscillatory shear stress stimulates adhesion molecule expression in
cultured human endothelium. Circ. Res. 82(5), 532-539 (1998)
102. Mugge, A., et al.: Release of intact endothelium-derived relaxing factor depends on
endothelial superoxide dismutase activity. Am. J. Physiol. 260(2 Pt 1), C219-C225 (1991)
103. Barger, A.C., et al.: Hypothesis: vasa vasorum and neovascularization of human coronary
arteries. A possible role in the pathophysiology of atherosclerosis. N. Engl. J. Med. 310(3),
175-177 (1984)
104. O'Brien, K.D., et al.: Neovascular expression of E-selectin, intercellular adhesion molecule-1,
and vascular cell adhesion molecule-1 in human atherosclerosis and their relation to intimal
leukocyte content. Circulation 93(4), 672-682 (1996)
105. De Keulenaer, G.W., et al.: Oscillatory and steady laminar shear stress differentially affect
human endothelial redox state: role of a superoxide-producing NADH oxidase. Circ. Res.
82(10), 1094-1101 (1998)
106. Ohara, Y., et al.: Lysophosphatidylcholine increases vascular superoxide anion production
via protein kinase C activation. Arterioscler. Thromb. 14(6), 1007-1013 (1994)
107. Heinloth, A., et al.: Stimulation of NADPH oxidase by oxidized low-density lipoprotein induces
proliferation of human vascular endothelial cells. J. Am. Soc. Nephrol. 11(10), 1819-1825 (2000)
108. Li, W.G., et al.: Activation of NAD(P)H oxidase by lipid hydroperoxides: mechanism of
oxidant-mediated smooth muscle cytotoxicity. Free Radic. Biol. Med. 34(7), 937-946 (2003)
109. Wolfort, R.M., Stokes, K.Y., Granger, D.N.: CD4+ T lymphocytes mediate
hypercholesterolemia-induced endothelial dysfunction via a NAD(P)H oxidase-dependent
mechanism. Am. J. Physiol. Heart Circ. Physiol. 294(6), H2619-H2626 (2008)
110. Moulton, K.S., et al.: Angiogenesis inhibitors endostatin or TNP-470 reduce intimal
neovascularization and plaque growth in apolipoprotein E-deficient mice. Circulation
99(13), 1726-1732 (1999)
111. Tenaglia, A.N., et al.: Neovascularization in atherectomy specimens from patients with
unstable angina: implications for pathogenesis of unstable angina. Am. Heart J. 135(1),
10-14 (1998)
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