Biomedical Engineering Reference
In-Depth Information
23. Winslow R, Greenstein J, Tomaselli G, O'Rourke B. 2001. Computational model of the failing
myocyte: Relating altered gene expression to cellular function.
Phil Trans Roy Soc Lond A
359
:1187-1200.
24. Hille B. 1992.
Ionic channels of excitable membranes
. Sinauer, Sundeland, MA.
25. Horn R, Vandenberg CA. 1984. Statistical properties of single sodium channels.
J Gen Physiol
84
(4):505-534.
26. Liu S, Rasmusson RL. 1997. Hodgkin-Huxley and partially coupled inactivation models yield
different voltage dependence of block.
Am J Physiol
272
(4, pt 2):H2013-H2022.
27. Irvine L. 1998.
Models of the cardiac Na channel and the action of lidocaine
. Department of
Biomedical Engineering. The Johns Hopkins University School of Medicine, Baltimore.
28. Irvine L, Jafri MS, Winslow RL. 1999. Cardiac sodium channel Markov model with tempera-
ture dependence and recovery from inactivation.
Biophys J
76
:1868-1885.
29. Clay JR, DeFelice LJ. 1983. Relationship between membrane excitability and single channel
open-close kinetics.
Biophys J
42
:151-157.
30. Corana A, Marchesi M, Martini C, Ridella S. 1987. Minimizing multimodal functions of con-
tinuous variables with the "simulated annealing" algorithm.
ACM Trans Math Software
13
(3):262-280.
31. Mazhari R, Greenstein JL, Winslow RL, Marban E, Nuss HB. 2001. Molecular interactions
between two long-QT syndrome gene products, HERG and KCNE2, rationalized by
in vitro
and
in silico
analysis.
Circ Res
89
(1):33-38.
32. Mazhari R, Nuss HB, Winslow RL, Marban E. 2002. Ectopic expression of KCNE3 in the
heart accelerated cardiac repolarization: a novel approach to gene therapy for long QT syn-
drome.
J Clin Invest
109
(8):1083-1090.
33. Greenstein J, Po S, Wu R, Tomaselli G, Winslow RL. 2000. Role of the calcium-independent
transient outward current Ito1 in action potential morphology and duration.
Circ Res
87
:1026.
34. Wagner J, Keizer J. 1994. Effects of rapid buffers on Ca2+ diffusion and Ca2+ oscillations.
Biophys J
67
(1).
35. Beuckelmann DJ, Nabauer M, Erdmann E. 1992. Intracellular calcium handling in isolated
ventricular myocytes from patients with terminal heart failure.
Circulation
85
:1046-1055.
36.
Kaab S, Nuss HB, Chiamvimonvat N, O'Rourke B, Pak PH, Kass DA, Marban E, Tomaselli
GF. 1996. Ionic mechanism of action potential prolongation in ventricular myocytes from dogs
with pacing-induced heart failure.
Circ Res
78
(2):262-273.
37.
O'Rourke B, Peng LF, Kaab S, Tunin R, Tomaselli GF, Kass DA, Marban E. 1999. Mecha-
nisms of altered excitation-contraction coupling in canine tachycardia-induced heart: experi-
mental studies.
Circ Res
84
:562-570.
38.
Beuckelmann DJ, Nabauer M, Erdmann E. 1993. Alterations of K
+
currents in isolated human
ventricular myocytes from patients with terminal heart failure.
Circ Res
73
:379-385.
39.
Näbauer M, Beuckelmann DJ, Überfuhr P, Steinbeck G. 1996. Regional differences in current
density and rate-dependent properties of the transient outward current in subepicardial and
subendocardial myocytes of human left ventricle.
Circulation
93
:168-177.
40.
Dixon JE, Shi W, Wang H-S, McDonald C, Yu H, Wymore RS, Cohen IS, McKinnon D. 1996.
Role of the Kv4.3 K+ Channel in Ventricular Muscle: A Molecular Correlate for the Transient
Outward Current.
Circ Res
79
:659-668.
41.
Kaab S, Dixon J, Duc J, Ashen D, Näbauer M, Beuckelmann DJ, Steinbeck D, McKinnon D,
Tomaselli GF. 1998. Molecular basis of transient outward potassium current downregulation in
human heart failure: a decrease in Kv4.3 mRNA correlates with a reduction in current density.
Circulation
98
:1383-1393.
42.
Takahashi T, Allen PD, Lacro RV, Marks AR, Dennis AR, Schoen FJ, Grossman W, Marsh JD
Izumo S. 1992. Expression of dihydropyridine receptor (Ca2+ channel) and calsequestrin genes
in the myocardium of patients with end-stage heart failure.
J Clin Invest
90
:927-935.
