Environmental Engineering Reference
In-Depth Information
WHO/FAO. 2010. Joint FAO/WHO expert meeting to review toxicological
and health aspects of bisphenol A and stakeholder meeting, Ottawa,
Canada, November 1-5, 2010. Geneva: World Health Organization.
Wong KO, Leo LW, Seah HL. Dietary exposure assessment of infants to
bisphenol A from the use of polycarbonate baby milk bottles. Food Addit
Contam 2005;22:280-288.
Wormuth M, Scheringer M, Vollenweider M, Hungerbuhler K. What are
the sources of exposure to eight frequently used phthalic acid esters in
Europeans? Risk Anal 2006;26:803-824.
Wozniak AL, Bulayeva NN, Watson CS. Xenoestrogens at picomolar to
nanomolar concentrations trigger membrane estrogen receptor-α mediated
Ca++ fluxes and prolactin release in GH3/B6 pituitary tumor cells. Environ
Health Perspect 2005;113:431-439.
Zalko D, Jacques C, Duplan H, Bruel S, Perdu E. Viable skin efficiently
absorbs and metabolizes bisphenol A. Chemosphere 2011;82 (3):424-430.
Zhou T, Ross DG, DeVito MJ, Crofton KM. Effects of short-term in vivo
exposure to polybrominated diphenyl ethers on thyroid hormones and
hepatic enzyme activities weanling rats. Toxicol Sci 2001;61:76-82.
Zhou T, Taylor MM, DeVito MJ, Crofton KM. Developmental exposure to
brominated diphenyl ethers results in thyroid hormone disruption. Toxicol
Sci 2002;66:105-116.
Zhou J, Yu Y, Chen D, Zhong Y. Effects of maternal oral feeding of
decabromodiphenyl ether (PBDE 209) on the humoral immunity of
offspring rats. J Trop Med (Guangzhou) 2010;10 (3):276-279.
Notes
1
The notion of EDCs is itself a recent one, and the term “endocrine
disruptor” was first used only in 1991 at the Wingspread Conference
Center in Racine, Wisconsin.
2
Thinning of eggshells in certain species of birds, the decrease in Baltic
Seal population, and adverse impacts on fish populations as a result of
exposure to EDCs have been reported.
