A Simple Method to Detect Allostery in GPCR Dimers - Methods in Cell Biology - page 173

De Lean, A., Stadel, J. M., & Lefkowitz, R. J. (1980). A ternary complex model explains
the agonist-specific binding properties of the adenylate cyclase-coupled b -adrenergic
receptor. The Journal of Biological Chemistry , 255 , 7108-7117.
Dupr´, D. J., & H´bert, T. E. (2006). Biosynthesis and trafficking of seven transmembrane
receptor signalling complexes. Cellular Signalling , 18 , 1549-1559.
Dupr´, D. J., Robitaille, M., Rebois, R. V., & H´bert, T. E. (2009). The role of G b g subunits in
the organization, assembly, and function of GPCR signaling complexes. Annual Review of
Pharmacology and Toxicology , 49 , 31-56.
Goupil, E., Tassy, D., Bourguet, C., Quiniou, C., Wisehart, V., P´trin, D., et al. (2010). A novel
biased allosteric compound inhibitor of parturition selectively impedes the prostaglandin
F2 a -mediated Rho/ROCK signaling pathway. The Journal of Biological Chemistry , 285 ,
25624-25636.
Guan, R., Feng, X., Wu, X., Zhang, M., Zhang, X., H´bert, T. E., et al. (2009). Biolumines-
cence resonance energy transfer studies reveal constitutive dimerization of the human
lutropin receptor and a lack of correlation between receptor activation and the propensity
for dimerization. The Journal of Biological Chemistry , 284 , 7483-7494.
Guan, R., Wu, X., Feng, X., Zhang, M., H ´ bert, T. E., & Segaloff, D. L. (2010). Structural
determinants underlying constitutive dimerization of unoccupied human follitropin recep-
tors. Cellular Signalling , 22 , 247-256.
Han, Y., Moreira, I. S., Urizar, E., Weinstein, H., & Javitch, J. A. (2009). Allosteric commu-
nication between protomers of dopamine class A GPCR dimers modulates activation.
Nature Chemical Biology , 5 , 688-695.
H ´ bert, T. E., & Bouvier, M. (1998). Structural and functional aspects of G protein-coupled
receptor oligomerization. Biochemistry and Cell Biology , 76 , 1-11.
Hlavackova, V., Goudet, C., Kniazeff, J., Zikova, A., Maurel, D., Vol, C., et al. (2005).
Evidence for a single heptahelical domain being turned on upon activation of a dimeric
GPCR. The EMBO Journal , 24 , 499-509.
Kamal, M., & Jockers, R. (2009). Bitopic ligands: All-in-one orthosteric and allosteric. F1000
Biology Reports , 1 , 77.
Kenakin, T. (2010). G protein coupled receptors as allosteric proteins and the role of allosteric
modulators. Journal of Receptor and Signal Transduction Research , 30 , 313-321.
Kenakin, T., &Miller, L. J. (2010). Seven transmembrane receptors as shapeshifting proteins:
The impact of allosteric modulation and functional selectivity on new drug discovery.
Pharmacological Reviews , 62 , 265-304.
Keov, P., Sexton, P. M., & Christopoulos, A. (2011). Allosteric modulation of G protein-
coupled receptors: A pharmacological perspective. Neuropharmacology , 60 , 24-35.
Kniazeff, J., Prezeau, L., Rondard, P., Pin, J. P., &Goudet, C. (2011). Dimers and beyond: The
functional puzzles of class C GPCRs. Pharmacology & Therapeutics , 130 , 9-25.
Kostenis, E., & Mohr, K. (1996). Two-point kinetic experiments to quantify allosteric effects
on radioligand dissociation. Trends in Pharmacological Sciences , 17 , 280-283.
Lanzafame, A., & Christopoulos, A. (2004). Investigation of the interaction of a putative
allosteric modulator, N-(2,3-diphenyl-1,2,4-thiadiazole-5-(2H)-ylidene) methanamine
hydrobromide (SCH-202676), with M1 muscarinic acetylcholine receptors. The Journal
of Pharmacology and Experimental Therapeutics , 308 , 830-837.
Lee, S. P., So, C. H., Rashid, A. J., Varghese, G., Cheng, R., Lanca, A. J., et al. (2004).
Dopamine D1 and D2 receptor Co-activation generates a novel phospholipase C-mediated
calcium signal. The Journal of Biological Chemistry , 279 , 35671-35678.