Organization of Genes Conferring Resistance to Anthracnose in Common Bean - Translational Genomics for Crop Breeding: Biotic Stress - page 153

(b)

(a) (c)

(d) (e)

Fig. 9.1. Anthracnose lesions on common bean leaf, seedling, pod, seed, and spores growing on
infected bean leaf on agar. For a color version of this figure, please refer to the color plate.

shrunken lesion on leaf veins, stems, and
hypocotyls of seedling plants. Lesions expand to
cause leaf flagging and wilting that exhibit typi-
cal chlorotic tissue not dissimilar to that observed
for other foliar pathogens. Infected plants pro-
duce infected seed and seed is the major means of
dispersal of the pathogen locally and across inter-
national boundaries and is the source of infection
next season. Plant debris infected with anthrac-
nose is not recognized as a major source of infec-
tion assuming farmers rotate crops, but infected

debris has survived 22 months in northern tem-
perate zones. Planting resistant cultivars is the
most effective control method.

Methods for Test Response to Pathogen

ResistanceTests

Plant diseases are a result of the interac-
tion between a susceptible host and a virulent
pathogen in favorable environmental conditions.