Biology Reference
In-Depth Information
Kanterewicz, B.I., Urban, N.N., McMahon, D.B.T., Norman, E.D., Giffen, L.J., Favata, M.F.,
Scherle, P.A., Trzăskos, J.M., Barrionuevo, G., and Klann, E. (2000). The extracellular
signal-regulated kinase cascade is required for NMDA receptor-independent LTP in area
CA1 but not area CA3 of the hippocampus. Journal of Neuroscience, 20, 3057-3066.
Kaplan, R.J., Greenwood, C.E., Winocur, G., and Wolever, T.M.S. (2000). Cognitive
performance is associated with glucose regulation in healthy elderly persons and can be
enhanced with glucose and dietary carbohydrates. American Journal of Clinical
Nutrition, 72, 825-836.
Kayed, R., Head, E., Thompson, J.L., McIntire, T.M., Milton, S.C., Cotman, C.W., and
Glabe, C.G. (2003). Common structure of soluble amyloid oligomers implies common
mechanism of pathogenesis. Science, 300, 486-489.
Kennedy, M.J., and Ehlers, M.D. (2006). Organelles and trafficking machinery for
postsynaptic plasticity. Annual Review of Neuroscience, 29, 325-362.
Kiselyov, V.V., Skladchikova, G., Hinsby, A.M., Jensen, P.H., Kulahin, N., Soroka, V.,
Pedersen, N., Tsetlin, V., Poulsen, F.M., Berezin, V., and Bock, E. (2003). Structural
basis for a direct interaction between FGFR1 and NCAM and evidence for a regulatory
role of ATP. Structure, 11, 691-701.
Klein, W.L. (2006). Synaptic targeting by Aβ oligomers (ADDLs) as a basis for memory loss
in early Alzheimer's disease. Alzheimer's and Dementia, 2, 43-55.
Knobloch, M., Konietzko, U., Krebs, D.C., and Nitsch, R.M. (2007a). Intracellular Aβ and
cognitive deficits precede β-amyloid deposition in transgenic arcAβ mice. Neurobiology
of Aging, 28, 1297-1306.
Knobloch, M., Farinelli, M., Konietzko, U., Nitsch, R.M., and Mansuy, I.M. (2007b). Aβ
oligomer-mediated long-term potentiation impairment involves protein phosphatase 1-
dependent mechanisms. Journal of Neuroscience, 27, 7648-7653.
Kokaia, Z., Andsberg, G., Yan, Q., and Lindvall, O. (1998). Rapid alterations of BDNF
protein levels in the rat brain after focal ischemia: evidence for increased synthesis and
anterograde axonal transport. Experimental Neurology, 154, 289-301.
Kondo, T., Shirasawa, T., Itoyama, Y., and Mori, H. (1996). Embryonic genes expressed in
Alzheimer's disease brains. Neuroscience Letters, 209, 157-160.
Korte, M., Carroll, P., Wolf, E., Brem, G., Thoenen, H. and Bonhoeffer, T. (1995).
Hippocampal long-term potentiation is impaired in mice lacking brain-derived
neurotrophic factor. Proceedings of the National Academy of Sciences, USA 92, 8856-
8860.
Korzus, E., Rosenfeld, M.G., and Mayford, M. (2004). CBP histone acetyltransferase activity
is a critical component of memory consolidation. Neuron, 42, 961-972.
Kotilinek, L.A., Bacskai, B., Westerman, M., Kawarabayashi, T., Younkin, L., Hyman, B.T.,
Younkin, S., and Ashe, K.H. (2002). Reversible memory loss in a mouse transgenic
model of Alzheimer's disease. Journal of Neuroscience, 22, 6331-6335.
Koudinov, A.R., and Koudinova, N.V. (2001). Essential role for cholesterol in synaptic
plasticity and neuronal degeneration. The FASEB Journal, 15, 1858-1860.
Kuwada, J.Y. (1986) Cell recognition by neuronal growth cones in a simple vertrebrate
embryo. Science, 233, 740-746.
Lacor, P.N., Buniel, M.C., Chang, L., Fernandez, S.J., Gong, Y., Viola, K.L., Lambert, M.P.,
Velasco, P.T., Bigio, E.H., Finch, C.E., Krafft, G.A., and Klein, W.L. (2004). Synaptic
Search WWH ::




Custom Search