Biology Reference
In-Depth Information
[15]
Anderson, P. et al. (2007).
Historical Perspective: Proposed Functions, Biological
Characteristics, and Neurobiological Models of the Hippocampus
, in
The Hippocampus
Book
, P. Anderson, et al., Editors. Oxford University Press: New York. p. 9-36.
[16]
Amaral, D., and Lavenex, P. (2007).
Hippocampal Neuroanatomy
, in
The Hippocampus
Book
, P. Anderson, et al., Editors. Oxford University Press: New York. p. 37-114.
[17]
Bear, M.F., and Malenka, R.C. (1994). Synaptic plasticity: LTP and LTD
.
Curr Opin
Neurobiol
, 4(3), p. 389-99.
[18]
Bear, M.F. (1995). Mechanism for a sliding synaptic modification threshold
.
Neuron
,
15(1), p. 1-4.
[19]
Albensi, B.C. et al. (2007). Electrical stimulation protocols for hippocampal synaptic
plasticity and neuronal hyper-excitability: are they effective or relevant?
Exp Neurol
,
204(1), p. 1-13.
[20]
Bliss, T., Collinridge, G., and Morris, R. (2007).
Synaptic plasticity in the
hippocampus
, in
The Hippocampus Book
, P. Anderson, et al., Editors. Oxford
University Press: New York. p. 343-474.
[21]
Martin, S.J., Grimwood, P.D., and Morris, R.G. (2000). Synaptic plasticity and
memory: an evaluation of the hypothesis
.
Annu Rev Neurosci
, 23, p. 649-711.
[22]
Otto, T. et al. (1991). Learning-related patterns of CA1 spike trains parallel stimulation
parameters optimal for inducing hippocampal long-term potentiation
.
Hippocampus
,
1(2), p. 181-92.
[23]
Whitlock, J.R. et al. (2006). Learning induces long-term potentiation in the
hippocampus.
Science
, 313(5790), p. 1093-7.
[24]
Pastalkova, E. et al. (2006). Storage of spatial information by the maintenance
mechanism of LTP
.
Science
, 313(5790), p. 1141-4.
[25]
Teyler, T.J. (1999). Use of brain slices to study long-term potentiation and depression
as examples of synaptic plasticity
.
Methods
, 18(2), p. 109-16.
[26]
Yang, S.N., Tang, Y.G., and Zucker, R.S. (1999). Selective induction of LTP and LTD
by postsynaptic [Ca2+]i elevation
.
J Neurophysiol
, 81(2), p. 781-7.
[27]
Kew, J.N. et al. (2000).
Functional consequences of reduction in NMDA receptor
glycine affinity in mice carrying targeted point mutations in the glycine binding site.
J
Neurosci, 20(11), p. 4037-49.
[28]
Watkins, J.C. (1994).
The NMDA receptor concept: origins and development
. 2nd ed.
The NMDA receptor, ed. Collingridge G.L., and Watkins. J.C. New York: Oxford
University Press.
[29]
Waxman, E.A., and Lynch, D.R. (2005). N-methyl-D-aspartate receptor subtypes:
multiple roles in excitotoxicity and neurological disease.
Neuroscientist
, 11(1), p. 37-
49.
[30]
Emptage, N.,. Bliss, T.V., and Fine, A. (1999). Single synaptic events evoke NMDA
receptor-mediated release of calcium from internal stores in hippocampal dendritic
spines
.
Neuron,
22(1), p. 115-24.
[31]
Wigstrom, H., and Gustafsson, B. (1986). Postsynaptic control of hippocampal long-
term potentiation.
J Physiol
(Paris), 81(4), p. 228-36.
[32]
Dingledine, R. et al. (1999). The glutamate receptor ion channels.
Pharmacol Rev
,
51(1), p. 7-61.
[33]
Nicoll, R.A., and Schmitz, D. (2005). Synaptic plasticity at hippocampal mossy fibre
synapses
.
Nat Rev Neurosci
, 6(11), p. 863-76.
