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SA regulated NPR1 independent defense mechanism (Desveaux et al. 2002). NPR1
mutants treated with exogenous SA, induced the sDNA binding of WHY1 protein of
Arabidopsis thaliana (Desveaux et al. 2004).
4   Conclusion
Cellular redox status is the imprint of ratio of reduced-to-non-reduced compounds
(Kornas et al. 2010 ) determined through the equilibrium of antioxidant system and
ROS (Gill and Tuteja 2010 ) regulating directly the cell metabolism. The role of
SA in controlling the interface of total cellular oxidation state and antioxidation
has been evidenced recently through research and literature. SA is not only shown
to regulate cell metabolism under abiotic and biotic stress conditions within the
systemic and par-systemically, but also during the synergy of two major causes of
stress, often met whenever disease epidemiology has been described. Nevertheless,
the role of abiotic stress in priming the biotic stress has been often suggested but
scarcely explained on the basis of regulatory affairs revealing molecular cause. SA
appears to be the potent plant hormone to regulate the defense signaling internally
under these conditions and may find its future prospects in modern agronomy to pro-
tect the crop at high time of disease incidence to favour enhanced growth and pro-
duction. The detailed research in this discipline and critical reviews therefore, are
awaited to update our understanding of the overlapping roles and regulations of SA.
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