Intracellular sensors of immunity and allogeneic hematopoietic stem cell transplantation - Immune Biology of Allogeneic Hematopoietic Stem Cell Transplantation - page 439

[70] Elmaagacli AH, Koldehoff M, Beelen DW. Improved outcome of hematopoietic SCT in
patients with homozygous gene variant of Toll-like receptor 9. Bone Marrow Transplant
2009;44(5):295-302.
[71] Bleakley M, Riddell SR. Molecules and mechanisms of the graft-versus-leukaemia
effect. Nat Rev Cancer 2004;4(5):371-80.
[72] Vincent K, Roy DC, Perreault C. Next-generation leukemia immunotherapy. Blood
2011;118(11):2951-9.
[73] van den Brink MR, Burakoff SJ. Cytolytic pathways in haematopoietic stem-cell trans-
plantation. Nat Rev Immunol 2002;2(4):273-81.
[74] Li H, Matte-Martone C, Tan HS, et al. Graft-versus-host disease is independent of in-
nate signaling pathways triggered by pathogens in host hematopoietic cells. J Immunol
2011;186(1):230-41.
[75] Franchi L, Park JH, Shaw MH, et al. Intracellular NOD-like receptors in innate immu-
nity, infection and disease. Cell Microbiol 2008;10(1):1-8.
[76] Mayor A, Martinon F, De Smedt T, Petrilli V, Tschopp J. A crucial function of SGT1 and
HSP90 in inflammasome activity links mammalian and plant innate immune respons-
es. Nat Immunol 2007;8(5):497-503.
[77] Gross O, Thomas CJ, Guarda G, Tschopp J. The inflammasome: an integrated view. Im-
munol Rev 2011;243(1):136-51.
[78] Galluzzi L, Vitale I, Abrams JM, et al. Molecular definitions of cell death subroutines:
recommendations of the Nomenclature Committee on Cell Death 2012. Cell Death
Differ 2012;19(1):107-20.
[79] Chen G, Shaw MH, Kim YG, Nunez G. NOD-like receptors: role in innate immunity and
inflammatory disease. Annu Rev Pathol 2009;4:365-98.
[80] Dinarello CA. Immunological and inflammatory functions of the interleukin-1 family.
Annu Rev Immunol 2009;27:519-50.
[81] Ting JP, Lovering RC, Alnemri ES, et al. The NLR gene family: a standard nomenclature.
Immunity 2008;28(3):285-7.
[82] Franchi L, Munoz-Planillo R, Nunez G. Sensing and reacting to microbes through the
inflammasomes. Nat Immunol 2012;13(4):325-32.
[83] Inohara N, Chamaillard M, McDonald C, Nunez G. NOD-LRR proteins: role in host-
microbial interactions and inflammatory disease. Annu Rev Biochem 2005;74:355-83.
[84] McDonald C, Inohara N, Nunez G. Peptidoglycan signaling in innate immunity and
inflammatory disease. J Biol Chem 2005;280(21):20177-80.
[85] Viala J, Chaput C, Boneca IG, et al. Nod1 responds to peptidoglycan delivered by the
Helicobacter pylori CAG pathogenicity island. Nat Immunol 2004;5(11):1166-74.
[86] Viala J, Sansonetti P, Philpott DJ. Nods and 'intracellular' innate immunity. C R Biol
2004;327(6):551-5.
[87] Hugot JP, Chamaillard M, Zouali H, et al. Association of NOD2 leucine-rich repeat
variants with susceptibility to Crohn's disease. Nature 2001;411(6837):599-603.
[88] Ogura Y, Bonen DK, Inohara N, et al. A frameshift mutation in NOD2 associated with
susceptibility to Crohn's disease. Nature 2001;411(6837):603-6.
[89] van Heel DA, Ghosh S, Butler M, et al. Muramyl dipeptide and toll-like receptor
sensitivity in NOD2-associated Crohn's disease. Lancet 2005;365(9473):1794-6.
[90] Miceli-Richard C, Lesage S, Rybojad M, et al. CARD15 mutations in Blau syndrome. Nat
Genet 2001;29(1):19-20.
[91] Maeda S, Hsu LC, Liu H, et al. Nod2 mutation in Crohn's disease potentiates NF-kap-
paB activity and IL-1beta processing. Science 2005;307(5710):734-8.
[92] Hysi P, Kabesch M, Moffatt MF, et al. NOD1 variation, immunoglobulin E and asthma.
Hum Mol Genet 2005;14(7):935-41.
[93] Martinon F, Burns K, Tschopp J. The inflammasome: a molecular platform trig-
gering activation of inflammatory caspases and processing of proIL-beta. Mol Cell
2002;10(2):417-26.
[94] Kovacsovics M, Martinon F, Micheau O, Bodmer JL, Hofmann K, Tschopp J. Overex-
pression of Helicard, a CARD-containing helicase cleaved during apoptosis, accelerates
DNA degradation. Curr Biol 2002;12(10):838-43.
[95] Boyden ED, Dietrich WF. Nalp1b controls mouse macrophage susceptibility to anthrax
lethal toxin. Nat Genet 2006;38(2):240-4.
[96] Franchi L, Eigenbrod T, Munoz-Planillo R, Nunez G. The inflammasome: a caspase-1-
activation platform that regulates immune responses and disease pathogenesis. Nat
Immunol 2009;10(3):241-7.
[97] Hawkins PN, Lachmann HJ, McDermott MF. Interleukin-1-receptor antagonist in the
Muckle-Wells syndrome. N Engl J Med 2003;348(25):2583-4.
[98] Saitoh T, Fujita N, Jang MH, et al. Loss of the autophagy protein Atg16L1 enhances
endotoxin-induced IL-1beta production. Nature 2008;456(7219):264-8.

444